Perception Test - Color Vision

Color Blindness Test

12 Ishihara-style plates generated programmatically. Each plate hides a number in a field of colored dots. Enter what you see - Or tap "None" if you see no number. This is a screening tool, not a clinical diagnosis.

8%
Males affected
0.5%
Females affected
4 types
Deficiency categories
12
Ishihara-style plates
🎨

Color Vision Screening

You will see 12 plates. For each, enter the number you see, or click "None". Take the test in good lighting. Remove any tinted glasses or contacts.

This is a screening tool only. A formal diagnosis requires examination by an optometrist or ophthalmologist.

How color vision works

Normal human color vision is trichromatic - It relies on three types of cone photoreceptors in the retina, each sensitive to different wavelengths of light. S-cones peak at ~420nm (short/blue), M-cones at ~530nm (medium/green), and L-cones at ~560nm (long/red). The brain combines signals from these three channels through opponent-process encoding: a red-green channel, a blue-yellow channel, and a light-dark channel. The peripheral retina, which underlies peripheral vision, relies on rod photoreceptors rather than cones and is unaffected by color blindness.

Color perception emerges from the ratios of cone responses, not their absolute activation levels - Which is why colors look roughly constant under different lighting conditions (color constancy). Deficiencies in one or more cone types alter how the brain encodes these ratios, making certain color pairs indistinguishable. This also explains why color blindness tests work by encoding numbers in hue contrast rather than lightness contrast - A person with normal trichromatic vision easily reads the number, while someone with a specific deficiency cannot distinguish the figure from its background.

Types of color blindness

TypeDefectColors confusedPrevalence (M)Severity
ProtanopiaL-cones absentRed/green1%Severe
ProtanomalyL-cones shiftedRed/orange1%Mild–mod
DeuteranopiaM-cones absentRed/green1.2%Severe
DeuteranomalyM-cones shiftedGreen/yellow5%Mild
TritanopiaS-cones absentBlue/yellow<0.1%Severe
AchromatopsiaAll cones absentAll colors<0.01%Complete

The Ishihara test

Dr. Shinobu Ishihara of the Imperial University of Tokyo introduced his pseudoisochromatic plate test in 1917, initially to screen Japanese army recruits. Over a century later, it remains the most widely used color vision test worldwide - Included in military entrance screenings, aviation medicals, and routine pediatric vision assessments in most countries.

Each Ishihara plate consists of a circular field of dots varying in color, size, and lightness. A number is embedded in dots of one hue against a background of dots in a confusable hue. Because the dots also vary in lightness and size, individuals cannot use brightness cues to identify the number - Only chromatic contrast works. The 38-plate standard edition includes demonstration plates (visible to everyone), screening plates (missed by color-deficient individuals), and diagnostic plates that differentiate specific deficiency types. The version in this test is programmatically generated to approximate the chromatic contrast of the standard plates and serves as a preliminary screening only. Because color rendering varies between displays, device choice can influence screen-based results - The same hardware effects documented in our mobile vs desktop reaction time comparison.

Who is affected? Genetics and prevalence

The most common forms of color blindness - Red-green deficiencies caused by mutations in the OPN1LW (L-cone) and OPN1MW (M-cone) genes - Are X-linked recessive traits. Because males have only one X chromosome, a single defective copy produces color blindness. Females have two X chromosomes, so both must carry the mutation - Making female color blindness approximately 16 times less common than male. An estimated 8% of males and 0.5% of females of Northern European ancestry have some form of red-green color deficiency.

Population groupPrevalence (males)Prevalence (females)
Northern European8%0.4%
East Asian5%0.3%
Sub-Saharan African3%0.2%
South Asian6.5%0.3%

Living with color blindness

For most people, color blindness is a mild inconvenience rather than a disability. Common adaptations include relying on position cues (traffic lights are always red-top, green-bottom), choosing careers that do not require color discrimination, and using digital accessibility tools. Browser extensions like Chrome's Color Enhancer and apps like Colorblindness Simulator help both those with color blindness and designers who want to create accessible content.

EnChroma glasses use a notch filter to enhance chromatic contrast for people with red-green deficiencies. While they do not restore normal trichromatic vision, they can meaningfully improve color discrimination for individuals with anomalous trichromacy (protanomaly, deuteranomaly). They are less effective for dichromats (protanopia, deuteranopia) and do not help with blue-yellow or total color blindness. If you scored poorly on this screening, consider discussing a formal evaluation with an optometrist. Caring for your vision is one part of overall cognitive wellbeing - see our evidence-based guide to brain health.

When to see an eye doctor

Congenital color blindness (present from birth) is stable throughout life and does not worsen or cause other vision problems. However, acquired color vision deficiency - Color blindness that develops in adulthood - Can be a sign of underlying conditions including glaucoma, macular degeneration, diabetic retinopathy, multiple sclerosis, or toxic reactions to medications such as hydroxychloroquine, ethambutol, or digoxin. If your color vision has changed noticeably in adulthood, this warrants prompt optometric evaluation.

Occupations with color vision standards - Commercial aviation, electrical work, law enforcement, and certain military roles - Require formal testing with calibrated Ishihara plates, Farnsworth D-15, or Nagel anomaloscope, not online screeners. This test can indicate whether further testing is warranted but cannot serve as a formal occupational screening. Always work with a qualified clinician for those determinations. For other vision-related cognitive measures, explore the Peripheral Vision test and the Anti-Saccade test.

Frequently Asked Questions - Color Blindness Test

What types of color blindness does this test detect?
The test is designed primarily for red-green deficiencies: deuteranopia (M-cone absent), deuteranomaly (M-cone shifted), protanopia (L-cone absent), and protanomaly (L-cone shifted). It is less sensitive for tritanopia (blue-yellow, rare). Achromatopsia (complete color blindness) would produce very low scores. Confirm any positive screen with an optometrist using calibrated plates.
Can screen settings cause false positives?
Yes - Blue light filters (Night Shift, f.lux), warm color temperature settings, and wide-gamut uncalibrated displays all shift the chromatic contrast of the plates. Disable all color correction software before taking this test. For other vision tests that are less screen-sensitive, see Peripheral Vision and Anti-Saccade.
Is acquired color blindness different from congenital color blindness?
Yes - Congenital color blindness is present from birth and stable for life. Acquired color vision deficiency develops in adulthood and can signal glaucoma, macular degeneration, multiple sclerosis, diabetic retinopathy, or medication toxicity (hydroxychloroquine, ethambutol). A change in color vision in adulthood warrants prompt optometric evaluation - This test cannot distinguish congenital from acquired deficiency.
Does color blindness affect performance on other tests on this platform?
Minimally. Stroop test uses named colors (the word "RED", "BLUE") so color discrimination is not required - The word meaning creates the conflict, not the hue. Pattern Recognition uses brightness and spatial contrast more than chromatic contrast, reducing the impact. Only the Color Blindness test itself is directly affected.